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TL;DR
The central result is that while oxidative stress is associated with ASD and selenoprotein dyshomeostasis may contribute to ASD etiology, current literature is variable and inconclusive.
Frontiers in Molecular Neuroscience · 3 authors, 3 centres
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The central result is that while oxidative stress is associated with ASD and selenoprotein dyshomeostasis may contribute to ASD etiology, current literature is variable and inconclusive.
The authors discuss evidence that oxidative stress is associated with ASD and that decreases in selenoproteins such as glutathiones have been observed in ASD patients, suggesting selenoprotein dyshomeostasis may play a role in ASD etiology.